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Generalized Epilepsy: Neurobiological Approaches ePub download

by AVOLI

  • Author: AVOLI
  • ISBN: 0817634452
  • ISBN13: 978-0817634452
  • ePub: 1399 kb | FB2: 1361 kb
  • Language: English
  • Category: Medicine & Health Sciences
  • Publisher: Birkhäuser; 1990 edition (January 1, 1990)
  • Pages: 482
  • Rating: 4.2/5
  • Votes: 212
  • Format: rtf lit mbr doc
Generalized Epilepsy: Neurobiological Approaches ePub download

In recent years there has been intense interest in the basic mechanisms of epilepsy. Generalized Epilepsy: Neurobiological Approaches.

In recent years there has been intense interest in the basic mechanisms of epilepsy. Many symposia and monographs have been devoted to this topic. It was natural that scientists interested in the fundamental mechanism of epileptogenesis at the cellular level were attracted to work on models in which the epileptogenic dysfunction could be confined to ever-smaller populations of neurons. Birkhäuser Boston, 1990.

Generalized Epilepsy. Neurobiological Approaches. In recent years there has been intense interest in the basic mechanisms of epilepsy. eBook 67,82 €. price for Russian Federation (gross).

Generalized Epilepsy: Neurobiological Approaches - Ebook written by AVOLI. Read this book using Google Play Books app on your PC, android, iOS devices. Download for offline reading, highlight, bookmark or take notes while you read Generalized Epilepsy: Neurobiological Approaches.

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Generalised Epilepsy: Neurobiological Approaches. Massachusetts: Birkhauser Boston, In. 1990:190–212. 81. Avoli, M, Gloor, P. The effects of transient functional depression ofthe thalamus on spindles and on bilateral synchronous epileptic discharges of feline generalised penicillin epilepsy. Epilepsia 1981;22:443–452.

Books on Epilepsy Home. Discount on medical books from Wisepress. Held at Sunlodge Hotel, Accra, Ghana - 16 - 22 January 2010 Organizers: Giuliano Avanzini and Marina Bentivoglio Local Organizers: Albert K Akpalu and Sammi K Ohene.

Generalized epilepsy is a form of epilepsy characterised by generalised seizures with no apparent cause. Generalized seizures, as opposed to focal seizures, are a type of seizure that impairs consciousness and distorts the electrical activity of the whole or a larger portion of the brain (which can be seen, for example, on hy, EEG).

cusses the neurobiological targets and basic aspects. evidence-based approach. Epilepsia 2013a; 54(1): 13-8.

Idiopathic generalized epilepsies (IGEs) constitute one third of all epilepsies. They are genetically determined and affect otherwise normal people of both sexes and all races. IGEs manifest with typical absences, myoclonic jerks, and generalized tonic-clonic seizures, alone or in varying combinations and severity. Absence status epilepticus (ASE) is common. Most syndromes of IGE start in childhood or adolescence, but some have an adult onset. They are usually lifelong, although a few are age-related. The EEG is the most sensitive test in the diagnosis and confirmation of IGE.

New books and E-books. This article is aimed at discussing the neurobiological and aspects of anxiolytic medications with special attention to epilepsy. Publishers : Journals. Bulletin Infirmier du Cancer. Dermato Mag. Environnement, Risques & Santé.

In recent years there has been intense interest in the basic mechanisms of epilepsy. Many symposia and monographs have been devoted to this topic. These reviews, however, have focused almost exclusively on studies performed in models of partial (or focal) epilepsy. It was natural that scientists interested in the fundamental mechanism of epileptogenesis at the cellular level were attracted to work on models in which the epileptogenic dysfunction could be confined to ever-smaller populations of neurons. Many of the most illuminating studies done in this field in the recent past have been carried out in vitro on brain slices or on dissociated neuronal cultures. Much valuable insight into the mechanisms of epileptogenesis has been obtained in this manner. One cannot, however, automatically assume that mechanisms elucidated in such exquisitely "focal" models are applicable to forms of epilepsy in which seizures from their very start involve the entire brain. One of the most difficult questions that arises in this context is how epileptogenic dysfunction, which can be understood in terms of mechanisms involving a few contiguous cells, can lead to what appears to be a simultaneous onset of seizure discharge in virtually all areas of the brain. Most of the workers who have made excellent contributions to the understanding of the cellular mechanisms involved in epileptogenesis have paid virtually no attention to this important problem.